View Products

Search Products

Order Form

Rejuvenation Medicine

FAQs

Health Concerns

Contact Us

Who We Are

Home

Combination of nutritional products designed to provide optimal quality and quantity of nutrients for daily use to enhance health and reduce the risk of illness.

It provides a broad –based easily assimilated multi-vitamin, multimineral with added highest quality full spectrum vitamin E, a vegetable and herbal concentrate, essential fatty acids, a highly absorbed calcium and a synergistic antioxidant formulation.

Multigenics Intensive Care without Iron
Unique E
Ultra Potent C
Phyto-complete
Ultra EPA-DHA
Cal Apatite with Magnesium/ Cal-Apatite Plus
Cyto-Redoxin

Vitamin E may limit stroke size

NEW YORK (Reuters) -- High levels of vitamin E in the diet may limit the amount of brain tissue affected by a stroke, reducing the injured area by as much as 50%, according to a study conducted in rats. Dr. H.B. van der Worp and colleagues at the University Hospital Utrecht in the Netherlands report their findings in the May issue of Stroke, published by the American Heart Association.

"Vitamin E is a vitamin that can also be called an antioxidant because it has the capacity to destroy certain chemicals that are naturally made by the body and that are known as oxidants," Dr. William I. Rosenblum, a pathologist at the Medical College of Virginia, Richmond, told Reuters Health.

"These chemicals can damage tissue either over long periods of time or, as some people think, over very short periods of time, such as when one might be having a stroke," he explained. "So by taking vitamin E supplements, it might be that one could build up your own protective mechanisms that would make it more difficult for these oxidizing substances to act and make the stroke worse."

In his editorial on the study, also published in Stroke, Rosenblum outlined the proposed mechanism whereby vitamin E limits stroke size by "scavenging" oxygen-centered free radicals from cells, thus reducing the concentrations of damaging free radical molecules in the cell.

"There is a lot of evidence that vitamin E is protective against heart attack and strokes," said Rosenblum.

The study by van der Worp and colleagues is based on tests of vitamin E-deficient rats, who had twice as much brain damage after a stroke than rats who had been fed diets containing larger than recommended amounts of the vitamin.

"Our results demonstrate that the amount of vitamin E in the... rodent diet provides substantial protection against focal cerebral ischemic damage (stroke)," write the researchers. The study results also suggest that vitamin E doses far higher than the 15 units per day recommended by the Food and Drug Administration may be beneficial, Rosenblum states, but more study is needed.

"No one really knows what a truly optimal amount of vitamin E would be," said Rosenblum. "I take 400 units a day every day. Based on what I've seen, that probably is a correct dose. I've seen studies where people took 50 or 100 units and it didn't seem to protect them. I think 400 units is really a happy medium that is enough to be useful and not enough to have any harm that anybody knows about."

SOURCE: Stroke 1998;29:1002-1006.

Vitamin E Prevents Early Plaque Formation
November 24, 1998.
Journal of the American Heart Association/MedscapeWire

In laboratory tests vitamin E prevented the early stages of plaque formation by preventing white blood cells from sticking to cells that line the artery wall another weapon in the antioxidant's attack on heart disease, according to research at UT Southwestern Medical Center at Dallas.

The study, the first to examine how vitamin E-enrichment of these white plaque-producing cells called monocytes affects adhesion to the cells that line arteries, was published in the November 24 edition of Circulation: Journal of the American Heart Association.

"This beneficial effect of vitamin E further strengthens its role as an adjunctive therapy in the management of atherosclerosis," said lead author Dr. Ishwarlal Jialal, a professor of pathology and internal medicine at UT Southwestern.

Scientists, including the UT Southwestern researchers, had already established that vitamin E can reduce susceptibility to atherosclerosis, or hardening of the arteries, because it inhibits the oxidation of low-density lipoprotein (LDL), or "bad" cholesterol. Two years ago, work by Jialal and Dr. Sridevi Devaraj, an instructor of pathology at UT Southwestern, showed the first intracellular effect of vitamin E * that it suppressed the function of monocytes.

The monocyte is the critical cell in early plaque development. An early stage of artery-clogging plaque involves the attachment of the monocyte to human endothelial cells the artery wall.

Preventing this step could be another important target in the treatment of atherosclerosis, said Jialal, who is also a senior investigator in the Center for Human Nutrition at UT Southwestern.

The laboratory study examined the effect that vitamin E had on the monocyte's ability to bind itself to endothelial cells.

"These and other studies support the concept that the possible beneficial effects of vitamin E supplementation in reducing coronary-artery disease can be attributed to its combined effects on inhibition of the oxidative modification of lipoproteins and its intracellular effects on cells critical in atherogenesis, such as monocytes," Jialal said.

While enrichment of monocytes with vitamin E decreased adhesion to endothelium, enrichment of both monocytes and endothelial cells resulted in greater inhibition of adhesion, the work showed.

"This is most likely what is occurring when one ingests vitamin E since it gets in all cell membranes," Jialal said.

This also represents the first demonstration that vitamin E has effects at the nuclear level on an important transcription factor. It inhibited the transcription factor NF-kappaB that is important for adhesion and inflammation. Jialal said they have elucidated the molecular events that cause vitamin E to decrease clogged arteries: It inhibits this transcription factor and decreases adhesion molecules, resulting in less adhesion of monocytes to the endothelium. These actions reduce plaque formation.

Humans can obtain this response by taking vitamin E supplements.

American diets may lack vitamin C
By Ann Quigley

NEW YORK, Aug 17 (Reuters) -- Arizona State University researchers found "surprisingly high rates" of vitamin C deficiency in a group of American middle class patients attending a healthcare facility for routine health exams.
Blood tests showed that 30% of 494 study participants were vitamin C depleted, and 6.3% suffered from the more severe condition, vitamin C deficiency, according to a report published in the Journal of the American College of Nutrition.

"If a physician examined these (vitamin C deficient) people," lead author Dr. Carol Johnston, told Reuters Health, "it is very likely they would have found some of the early indications of scurvy." Scurvy, a disease caused by vitamin C deficiency, is thought to be rare in modern societies with year-round access to fresh fruits and vegetables. The early symptoms of scurvy include tiredness, weakness, irritability, weight loss, and vague muscle aches.

Although the researchers did not obtain dietary data on the study subjects, those who were vitamin C deficient and vitamin C depleted were probably not eating enough fruits and vegetables that are high in vitamin C, said Johnston. Fresh produce with high levels of the vitamin include oranges, grapefruits, broccoli, cauliflower, and cabbage.

"If you are just eating apples, bananas, green beans and lettuce, that's not going to do it," said Johnston. "They are the most commonly consumed and they literally have no vitamin C. Potatoes have a little bit. I think french fries are probably keeping people over the edge of having a problem."

In their report, the researchers also note that the "poor vitamin C status of some of our subjects may be related in part to smoking." Previous studies have shown that smokers need over 200 milligrams of the vitamin daily to maintain blood levels comparable to those of nonsmokers consuming the recommended daily allowance of the vitamin.

"Efforts should be made to further establish the prevalence of poor vitamin C status in the US population and to delineate the clinical significance of below normal plasma vitamin C concentrations," conclude the research team.

SOURCE: Journal of the American College of Nutrition 1998;17:366-370.